Broken Capillaries on Face: Causes, Treatments, and Prevention
Key points
- Genetics: A significant factor, with some studies suggesting up to 90% of people with spider veins have a family history of the condition. You may simply inherit weaker, more fragile blood vessel walls. Specific genetic variations can affect the production of structural proteins like collagen and elastin, as well as influence how blood vessels respond to temperature fluctuations and inflammatory mediators. Individuals with lighter skin tones and those of Northern European descent often notice vascular changes earlier simply because lower melanin levels provide less optical masking of underlying vasculature.
- Rosacea: This chronic inflammatory skin condition is a primary culprit. The persistent flushing and inflammation associated with rosacea can, over time, cause capillaries to dilate permanently. In rosacea patients, neurovascular dysregulation leads to exaggerated blood vessel responses to stimuli like spicy foods, stress, and skincare actives. The repeated cycles of flushing and cooling eventually stretch the elastic fibers around the capillaries until they remain permanently open. Subtypes 1 (erythematotelangiectatic) and 2 (papulopustular) are particularly associated with visible telangiectasia.
- Hormonal Fluctuations: While not always immediately obvious, hormones play a massive role in vascular tone. Elevated estrogen levels during pregnancy, from oral contraceptive use, or during menopause can cause blood vessels to relax and dilate. The condition often peaks in women during their 30s to 50s, coinciding with natural hormonal transitions. Increased blood volume during pregnancy also places additional pressure on delicate facial vessels, contributing to their expansion.
- Aging: As we age, our skin produces less collagen—the protein that provides structure and support. This, combined with thinning skin, makes blood vessels more fragile and visible, as noted by Columbia Skin Clinic. Natural aging processes also reduce subcutaneous fat in the face, removing a layer of cushioning that previously hid superficial vasculature.
Those fine, reddish lines that seem to have taken up permanent residence on your cheeks or around your nose are a common cosmetic concern known as broken capillaries. While generally harmless, their appearance can be frustrating. Fortunately, understanding what causes them is the first step toward effective treatment and prevention.
This comprehensive guide synthesizes information from leading dermatology clinics, medical resources, and user experiences to cover the causes, professional treatments, and preventative strategies for managing broken capillaries on the face. We will also explore the underlying vascular anatomy, differentiate between common skin conditions, and provide actionable advice for maintaining long-term vascular health. Whether you are noticing early signs of facial redness or dealing with established spider veins, this resource will help you navigate your options with confidence and clarity.
What Are Broken Capillaries?
Broken capillaries, medically termed telangiectasia and often called "spider veins," are not actually broken but rather permanently dilated or enlarged blood vessels located just beneath the skin's surface. This enlargement makes them visible as fine red, purple, or blue lines that can appear as single strands, small clusters, or web-like patterns.
To understand why they become visible, it helps to review basic facial skin anatomy. The superficial layers of the dermis contain a rich network of tiny blood vessels known as the papillary dermal plexus. These capillaries deliver oxygen and nutrients to the skin while removing metabolic waste. Normally, the surrounding collagen and elastin fibers act as structural scaffolding, keeping the vessel walls taut and compact. When this supportive matrix weakens due to aging, UV exposure, or chronic inflammation, the vessel walls lose their natural tone and begin to stretch. Once a vessel dilates beyond approximately 0.1 to 0.3 millimeters in diameter, it becomes optically visible through the epidermis. The "broken" terminology is a misnomer that stuck over decades because the sudden appearance of these vessels often mimics the look of a bruised or ruptured vein, but in reality, the vessel remains fully intact, just abnormally widened.
According to Medical News Today, they most frequently appear in delicate areas like the face—particularly the cheeks, nose, and chin—and are a common issue for adults of all ages, though some people are more susceptible than others. The skin on the face is inherently thinner than skin on the rest of the body, particularly in the periorbital and nasal regions, which naturally increases vascular visibility. Additionally, the face is constantly exposed to environmental stressors, making it the primary site for telangiectasia development. Understanding this anatomical context is crucial for setting realistic expectations about both prevention and professional intervention.
Common Causes: Why Do I Have Broken Capillaries?
The development of broken capillaries is often multifactorial, stemming from a combination of genetic predispositions and external triggers. Dermatologists typically classify these triggers into intrinsic (internal) and extrinsic (external) categories. Identifying your specific triggers is essential, as eliminating ongoing exposure can significantly slow progression and improve treatment outcomes.
Genetic and Intrinsic Factors
- Genetics: A significant factor, with some studies suggesting up to 90% of people with spider veins have a family history of the condition. You may simply inherit weaker, more fragile blood vessel walls. Specific genetic variations can affect the production of structural proteins like collagen and elastin, as well as influence how blood vessels respond to temperature fluctuations and inflammatory mediators. Individuals with lighter skin tones and those of Northern European descent often notice vascular changes earlier simply because lower melanin levels provide less optical masking of underlying vasculature.
- Rosacea: This chronic inflammatory skin condition is a primary culprit. The persistent flushing and inflammation associated with rosacea can, over time, cause capillaries to dilate permanently. In rosacea patients, neurovascular dysregulation leads to exaggerated blood vessel responses to stimuli like spicy foods, stress, and skincare actives. The repeated cycles of flushing and cooling eventually stretch the elastic fibers around the capillaries until they remain permanently open. Subtypes 1 (erythematotelangiectatic) and 2 (papulopustular) are particularly associated with visible telangiectasia.
- Hormonal Fluctuations: While not always immediately obvious, hormones play a massive role in vascular tone. Elevated estrogen levels during pregnancy, from oral contraceptive use, or during menopause can cause blood vessels to relax and dilate. The condition often peaks in women during their 30s to 50s, coinciding with natural hormonal transitions. Increased blood volume during pregnancy also places additional pressure on delicate facial vessels, contributing to their expansion.
- Aging: As we age, our skin produces less collagen—the protein that provides structure and support. This, combined with thinning skin, makes blood vessels more fragile and visible, as noted by Columbia Skin Clinic. Natural aging processes also reduce subcutaneous fat in the face, removing a layer of cushioning that previously hid superficial vasculature.
Environmental and Sun Damage
Excessive and cumulative sun exposure is one of the most significant preventable causes. UV radiation damages collagen and weakens the walls of blood vessels, making them more prone to dilation. As explained by Mayo Clinic, sun damage is a major contributor to the breakdown of skin elasticity and vessel integrity. Specifically, UVA rays penetrate deeply into the dermis, generating reactive oxygen species (ROS) that degrade vascular support structures. Over time, this chronic photoaging leads to a condition known as poikiloderma of Civatte, which combines telangiectasia, hyperpigmentation, and skin thinning primarily on the sides of the neck and lower cheeks. Even incidental daily sun exposure, like driving or walking to work, accumulates and contributes to gradual vessel dilation if proper photoprotection is neglected.
Lifestyle Choices
- Alcohol Consumption: Alcohol temporarily dilates blood vessels. Frequent or excessive consumption can lead to prolonged dilation and the formation of spider veins. While moderate drinking rarely causes permanent damage on its own, heavy or chronic alcohol intake leads to sustained vasodilation and can exacerbate underlying rosacea. It also contributes to systemic inflammation and dehydrates the skin, compromising barrier function.
- Smoking: Smoking damages blood vessels throughout the body, impairing their function and elasticity, which can contribute to their appearance on the face. The nicotine and carbon monoxide in cigarette smoke cause intermittent vasoconstriction followed by rebound vasodilation. This constant stress on vessel walls weakens their structural integrity over time. Smoking also reduces oxygen delivery to the skin and severely degrades collagen production.
- Dietary Triggers: For some individuals, highly spiced foods, very hot beverages, and foods rich in histamine or tyramine can trigger neurovascular flushing. Repeated episodes of intense facial flushing, regardless of the trigger, mechanically stress capillary walls and accelerate permanent dilation.
Physical Trauma and Pressure
- Skin Trauma: Harsh scrubbing, aggressive extractions, or even popping a pimple too forcefully can damage delicate capillaries. The mechanical pressure of manual exfoliation, rough cleansing brushes, or improper use of comedone extractors creates localized inflammation and physical disruption to the superficial vascular plexus. Dermatologists strongly advise against high-friction skincare tools for those prone to telangiectasia.
- Sudden Pressure Changes: An intense sneeze, cough, or bout of vomiting can create a sudden spike in pressure that causes small vessels in the face to burst. These acute pressure surges are typically harmless and often resolve spontaneously, but they can leave behind dilated capillaries in susceptible individuals. Similar mechanisms apply to intense weightlifting or straining, where the Valsalva maneuver increases venous pressure in the head and neck.
- Extreme Temperatures: Exposure to very hot environments (like saunas or hot showers) or extreme cold and wind can cause rapid expansion and contraction of blood vessels, weakening them over time. This repeated thermal cycling is particularly damaging to facial skin, which lacks the protective clothing and thicker subcutaneous fat found elsewhere. Chronic windburn causes micro-inflammation and barrier disruption, making capillaries more reactive to subsequent stimuli.
- Medication-Induced Dilation: Long-term use of topical or oral corticosteroids is a well-documented cause of facial telangiectasia. Steroids inhibit collagen synthesis and thin the dermal layer, exposing and weakening underlying vessels. Blood pressure medications, vasodilators, and certain hormone replacement therapies can also contribute to persistent facial redness and visible capillary networks.
Professional Treatments: How to Get Rid of Broken Capillaries
While topical products can help manage redness, professional dermatological treatments are the only way to effectively eliminate existing broken capillaries. These procedures rely on selective photothermolysis, a principle where specific wavelengths of light target a particular chromophore (in this case, oxyhemoglobin in blood) without damaging surrounding tissues. When selecting a treatment, board-certified dermatologists consider vessel diameter, depth, skin tone, and patient medical history to optimize outcomes and minimize risks like hyperpigmentation or scarring.
Laser Therapy
Laser treatments are widely considered the gold standard. They work by sending a concentrated beam of light that is absorbed by the hemoglobin in the blood. This energy converts to heat, which coagulates the blood and collapses the vessel wall. Once the vessel wall is destroyed, the body's lymphatic system gradually clears away the coagulated debris over the following weeks, and the visible line disappears.
- Pulsed Dye Laser (PDL): Often cited as the most effective, especially the V-beam laser, which specifically targets the red pigment in blood vessels without damaging surrounding skin. The PDL operates at a wavelength of 585 or 595 nanometers, which aligns perfectly with the absorption peak of hemoglobin. It is highly effective for fine, red, superficial telangiectasias and is frequently the first-line treatment for rosacea-associated vascular lesions. Modern PDL devices incorporate dynamic cooling systems that spray cryogen onto the skin milliseconds before the laser pulse, protecting the epidermis and significantly reducing discomfort and post-treatment downtime.
- Nd:YAG Laser: Effective for targeting deeper or larger blue-toned vessels. Operating at 1064 nm, this wavelength penetrates deeper into the dermis with less absorption by melanin, making it safer for darker skin tones. It is particularly useful for treating larger, more resistant facial veins that have a blue or purple hue, as well as telangiectasias around the nose where vessels sit slightly deeper.
- KTP Laser: The 532 nm wavelength is highly absorbed by hemoglobin, making it excellent for extremely fine, superficial red vessels. It is often used in combination with other lasers to treat different layers of the vascular plexus simultaneously.
Intense Pulsed Light (IPL)
IPL, also known as a "photofacial," uses broad-spectrum light to target redness and pigmentation. Unlike a laser, which uses a single wavelength, IPL emits multiple wavelengths of light. This makes it highly effective for treating widespread redness and numerous small capillaries, as seen in rosacea. While IPL is generally less powerful than targeted lasers, its advantage lies in treating larger surface areas with fewer passes, addressing both vascular and pigmentary concerns in a single session. It is particularly beneficial for patients with photoaging, sun damage, and diffuse facial erythema rather than isolated, prominent spider veins.
Pre-Treatment Considerations and Patient Selection
Not everyone is an immediate candidate for vascular laser therapy. Patients must typically avoid sun exposure, tanning beds, and self-tanners for at least four to six weeks prior to treatment, as tanned skin increases the risk of epidermal burns and post-inflammatory hyperpigmentation. Certain medications, such as isotretinoin (within the last 6-12 months depending on provider protocol) and photosensitizing antibiotics, require a washout period. A thorough consultation will include a review of your medical history, skin type assessment using the Fitzpatrick scale, and a discussion of realistic outcomes. In-office numbing creams are routinely applied to ensure patient comfort, as the laser pulse often feels like a quick rubber band snap followed by mild warmth.
Comparative Efficacy of Professional Treatments
| Treatment | Mechanism | Best For | Sessions Needed (Avg.) |
|---|---|---|---|
| Laser Therapy | Focused light beam collapses vessel | Individual, prominent, or deeper veins | 2-4 |
| IPL / BBL | Broad-spectrum light destroys vessel | Widespread redness, small superficial veins | 3-5 |
| Sclerotherapy | Injected solution collapses vessel | Larger, more prominent facial veins | 1-3 |
| Electrosurgery | Electrical current coagulates vessel | Very fine, isolated veins | 1-2 |
Recovery from these treatments typically involves a few days of mild redness or swelling. Results appear gradually over 2-6 weeks as the body absorbs the treated vessels.
Following laser or IPL treatment, patients may experience temporary perivascular edema, mild crusting, or a phenomenon known as "purpura," where treated vessels turn dark purple or brown before fading. Strict sun protection and gentle skincare are mandatory during this healing window. Most practitioners recommend avoiding vigorous exercise, hot showers, saunas, and alcohol for at least 48 hours to prevent reactive flushing. Maintenance sessions are typically required every 12 to 24 months, as new capillaries can form due to aging, lifestyle, or untreated underlying conditions like rosacea. Combining vascular lasers with topical anti-inflammatory regimens often yields the most comprehensive and long-lasting aesthetic results.
The Role of Skincare and Home Remedies
Setting Realistic Expectations
It is crucial to understand that topical creams, serums, and home remedies cannot eliminate existing broken capillaries. Their primary role is to strengthen the skin barrier, reduce inflammation and redness, and help prevent new ones from forming. Once a capillary has permanently dilated, no amount of topical application can shrink it back to its original size. Skincare should be viewed as a supportive therapy that optimizes skin health, reduces baseline erythema, and protects the results of professional procedures.
Key Skincare Ingredients
- Retinoids (Retinol, Tretinoin): By stimulating collagen production, retinoids can thicken the skin, making underlying vessels less visible. Prescription tretinoin and over-the-counter retinoids accelerate epidermal turnover and boost dermal glycosaminoglycan synthesis. However, they can initially cause irritation and transient redness, so patients prone to telangiectasia should introduce them slowly, starting with low concentrations and buffering them with moisturizer.
- Vitamin C: A powerful antioxidant that helps build collagen and can strengthen capillary walls. Topical L-ascorbic acid neutralizes free radicals generated by UV exposure and environmental pollutants, protecting the vascular support matrix. It also has mild brightening properties that can optically counteract diffuse redness when used consistently.
- Niacinamide (Vitamin B3): An anti-inflammatory ingredient that strengthens the skin barrier and can reduce overall redness. At concentrations of 4-10%, niacinamide calms neurogenic inflammation, improves ceramide synthesis, and modulates the skin's immune response. It is exceptionally well-tolerated and pairs beautifully with other actives without triggering the reactive flushing associated with stronger acids.
- Vitamin K: Often recommended post-procedure, topical Vitamin K may help reduce the appearance of redness and bruising. It is believed to assist in the coagulation cascade and support vascular integrity, though clinical evidence for its efficacy on established telangiectasia remains limited. It is most beneficial in soothing post-laser erythema and supporting skin recovery.
- Centella Asiatica & Licorice Root Extract: These botanical anti-inflammatories are increasingly featured in vascular-calming formulations. They work by inhibiting pro-inflammatory cytokines and strengthening capillary walls through improved microcirculation. They are ideal for patients with sensitive, reactive skin who cannot tolerate traditional retinoids or high-strength acids.
Building a Vascular-Friendly Routine
A successful skincare regimen for capillary-prone skin focuses on barrier integrity, gentle cleansing, and consistent photoprotection. Avoid products with high concentrations of alcohol, fragrant essential oils, menthol, eucalyptus, or physical scrubs, as these can trigger neurovascular reactivity. Use lukewarm water for cleansing, apply products with light patting motions rather than rubbing, and always finish with a barrier-repair moisturizer containing ceramides, squalane, or hyaluronic acid. For immediate cosmetic improvement, green-tinted color-correcting primers or mineral foundations containing iron oxides and zinc oxide can neutralize redness while providing gentle sun protection and physical coverage that is less likely to irritate reactive vessels.
Prevention: The Best Long-Term Strategy
Since recurrence is common, prevention is the most important part of managing broken capillaries. A proactive, multi-layered approach addressing environmental, behavioral, and physiological factors will significantly delay the formation of new telangiectasias and preserve the results of professional treatments.
- Sun Protection is Non-Negotiable: Daily application of a broad-spectrum sunscreen with an SPF of 30 or higher is the single most effective step you can take. Opt for mineral-based sunscreens containing zinc oxide or titanium dioxide, which reflect UV radiation and are less likely to cause stinging or heat-induced flushing compared to some chemical filters. Reapply every two hours during prolonged outdoor exposure, and consider sun-protective clothing like UPF-rated wide-brimmed hats.
- Practice Gentle Skincare: Avoid harsh scrubs, abrasive tools, and steaming hot water. Cleanse your face gently with lukewarm water and pat dry. Replace washcloths with ultra-soft silicone cleansing pads or simply use your fingertips. When exfoliating, stick to low-percentage polyhydroxy acids (PHAs) or enzymatic peels once a week rather than daily retinoids or glycolic acid, which can compromise the barrier in vascular-sensitive individuals.
- Avoid Temperature Extremes: Protect your face with a scarf in cold, windy weather and avoid overly hot showers and saunas. When transitioning from cold outdoor air to heated indoor environments, allow your skin to acclimate gradually rather than subjecting it to immediate high heat. Consider using a humidifier during winter months to prevent indoor heating from drying and irritating the facial skin barrier.
- Mindful Lifestyle Habits: Limit alcohol consumption and quit smoking to protect the health and integrity of your blood vessels. Maintain adequate hydration to support cellular function and skin elasticity. Incorporate a diet rich in bioflavonoids (found in citrus fruits, berries, and leafy greens) and omega-3 fatty acids, which support microvascular health and reduce systemic inflammation. Regular moderate exercise improves overall circulation without the extreme pressure spikes associated with heavy weightlifting or high-intensity interval training, though consulting your physician about appropriate exercise routines is always recommended.
- Manage Underlying Conditions: If you have rosacea, working with a dermatologist to control flare-ups is essential to prevent the formation of new telangiectasias. This may involve prescription topical therapies like ivermectin, brimonidine, or azelaic acid, as well as trigger tracking to identify and avoid personal dietary or environmental aggravators. Stress management through mindfulness, adequate sleep, and regulated breathing exercises can also blunt the sympathetic nervous system responses that drive facial flushing.
When to See a Dermatologist
You should consult a board-certified dermatologist if:
- You are bothered by the cosmetic appearance and wish to explore treatment options.
- You notice a sudden increase in the number of broken capillaries.
- The redness is accompanied by other symptoms like persistent flushing, bumps, or swelling.
Additionally, seek medical evaluation if you develop telangiectasia in atypical patterns or alongside systemic symptoms such as unexplained joint pain, gastrointestinal issues, or frequent nosebleeds. In rare cases, widespread or atypical telangiectasia can be a cutaneous marker of underlying systemic conditions, such as hereditary hemorrhagic telangiectasia (HHT), liver disease, or connective tissue disorders like lupus. A dermatologist can perform a thorough clinical examination, potentially utilizing dermoscopy to differentiate true vascular dilation from conditions like erythema, hyperpigmentation, or early skin cancers like basal cell carcinoma, which can sometimes present with arborizing vessels.
During a consultation, your dermatologist will map out a personalized treatment plan, discuss the expected number of sessions, outline post-procedure care, and provide guidance on insurance coverage. While treatments for broken capillaries are generally classified as cosmetic and not covered by standard health insurance, some cases involving functional rosacea or symptomatic vascular malformations may qualify for partial coverage depending on your specific plan and regional regulations. Understanding your diagnostic status and treatment roadmap empowers you to make informed decisions about both your skin health and your financial investment.
By combining professional treatments with a preventative skincare and lifestyle approach, you can effectively manage broken capillaries for a clearer, more even-toned complexion.
Disclaimer: This article is for informational purposes only and does not constitute medical advice. Always consult with a qualified healthcare professional for any health concerns or before making any decisions related to your health or treatment.
Frequently Asked Questions
Do broken capillaries on the face ever go away on their own?
Once a facial capillary has permanently dilated, it will not spontaneously revert to its original size or disappear without professional intervention. The structural proteins supporting the vessel wall have been compromised, resulting in a fixed, widened state. While baseline redness from temporary flushing may fade as the underlying inflammation subsides, the distinct thread-like lines of telangiectasia remain stable unless treated with targeted therapies like lasers or IPL. Consistent skincare and sun protection can prevent them from worsening, but removal requires clinical treatment.
Can I safely use at-home IPL devices to treat facial capillaries?
At-home IPL devices are significantly less powerful than professional medical-grade systems and generally cannot deliver the concentrated energy required to permanently collapse established facial telangiectasia. While they may provide mild, temporary improvements in overall diffuse redness, they carry a higher risk of adverse effects if used improperly, including burns, hyperpigmentation, and paradoxical irritation of sensitive vessels. Additionally, the broader beam and lack of precision cooling mechanisms make it difficult to safely target individual capillaries without affecting surrounding skin. Dermatologists typically recommend reserving IPL for in-office use where parameters can be precisely calibrated to your skin type and vessel depth.
Is there a connection between broken capillaries and high blood pressure?
Hypertension and facial telangiectasia share a relationship with vascular stress, but they are not directly causative in the way many assume. Systemic high blood pressure primarily affects larger arteries and arterioles rather than the microscopic capillaries near the skin's surface. However, medications used to treat hypertension, particularly vasodilators like calcium channel blockers, can sometimes cause persistent facial flushing and exacerbate the visibility of existing capillaries. If you are managing hypertension and notice a sudden proliferation of facial spider veins, it is advisable to discuss medication side effects with your physician rather than assuming the blood pressure itself is causing the visible skin changes.
How can I tell the difference between rosacea and simply having broken capillaries?
Broken capillaries (telangiectasia) are a symptom, whereas rosacea is a chronic inflammatory condition that often causes them. If your primary concern is isolated, fine red lines without persistent background redness, burning, stinging, or acne-like bumps, you likely have standalone telangiectasia driven by sun exposure, genetics, or aging. Rosacea, however, typically presents with central facial erythema, episodic flushing, visible capillaries, and often papules or pustules, frequently accompanied by skin sensitivity or ocular irritation. Many patients have both, as rosacea inflammation accelerates capillary dilation. A dermatologist can differentiate the two using clinical history, dermoscopic examination, and response to targeted therapies, ensuring your treatment addresses the root cause rather than just the surface symptom.
Will drinking more water or applying ice fix broken capillaries?
Hydration is essential for overall skin barrier function and cellular health, and drinking adequate water can improve skin plumpness and elasticity. However, it will not reverse permanently dilated capillaries. Similarly, while applying ice or cold compresses can induce temporary vasoconstriction, reducing redness and puffiness for a few minutes, it does not repair the damaged vessel wall or permanently shrink the telangiectasia. In fact, extreme temperature changes—including frequent icing—can sometimes trigger reactive flushing or compromise the skin barrier if overdone. Ice should only be used briefly for soothing purposes, not as a therapeutic treatment for vascular removal. Sustainable results require professional intervention and consistent protective habits.
Conclusion
Broken capillaries, or facial telangiectasia, represent a common and highly treatable vascular concern driven by a complex interplay of genetics, environmental exposure, aging, and lifestyle factors. Understanding that these vessels are dilated rather than ruptured helps clarify why topical products alone cannot eliminate them, though they play a vital supportive role in strengthening the skin barrier and reducing baseline redness. Professional interventions such as pulsed dye lasers, Nd:YAG systems, and IPL remain the most effective methods for permanently clearing visible capillary networks, with modern technology offering safer, more comfortable, and highly customized treatments for all skin tones.
Long-term management relies heavily on proactive prevention. Strict daily sun protection, gentle cleansing routines, avoidance of thermal extremes, and mindful management of dietary or lifestyle triggers are non-negotiable for preserving treatment results and preventing recurrence. Patients with underlying conditions like rosacea should prioritize comprehensive inflammatory control alongside vascular treatments. By partnering with a board-certified dermatologist to develop an individualized plan, setting realistic expectations regarding maintenance, and adhering to evidence-based skincare principles, individuals can successfully minimize the appearance of broken capillaries and maintain a resilient, evenly toned complexion for years to come.
About the author
Elena Vance, MD, is a double board-certified dermatologist and pediatric dermatologist. She is an assistant professor of dermatology at a leading medical university in California and is renowned for her research in autoimmune skin disorders.